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Breakthrough infections can lead to long Covid
Vaccines Keep You Safe From the Fatal Upsets but Not From the Lingering Hustle
What the Latest Study Says
Quick Takeaways
Why It Matters
“Long COVID and other lingering health bumps are a real head‑ache for millions,” the researchers explained. “These problems can stick around, causing lasting distress, so it’s a worry that vaccines don’t fully guard against them.”
Bottom Line in Plain English
Think of vaccination as a bodyguard who stops the strongest villains from getting in, but it isn’t your personal healing therapist for the post‑battle aftermath.
Genes may explain critical Covid-19 in young, healthy adults
Why a Gene Called ADAM9 Might Be the Dark Horse Behind Severe Covid in Young, Healthy Folks
Picture this: You’re under 50, you’ve never had a heart attack, a stroke, or diabetes—yet you’re pushing through a hospital bed because Covid won’t quit. A team of French scientists has stumbled upon a surprising culprit in the mix:
- Neatly identified five genes that were “upregulated” (boosted) in patients with critical Covid.
- The star of the show? The gene known as ADAM9.
They did the numbers right: 72 patients, none with pre‑existing chronic conditions known to doom Covid outcomes, and a control group of 22 healthy volunteers. Even more intriguing, a separate cohort of 154 patients reproduced the same genetic pattern—deemed a robust signal, not a one‑time fluke.
What ADAM9 Does Under the Hood
In a lab set‑up that mimics the human lung, scientists tossed a genetic “block” onto ADAM9 and watched the virus’s replication engines grind to a halt. Imagine a factory that suddenly loses its right-hand robot—production slows dramatically.
Why It Matters
If ADAM9 helps the virus clone itself, blocking it could be the next trick in the vaccine or therapeutic toolbox. Think of it as a “silencer” to the virus’s assembly line.
The Road Ahead
- More studies are needed to confirm these findings.
- Researchers will look into whether targeting ADAM9 is worthwhile…
- Will it be a quick fix for the next outbreak, or just a footnote in Covid lore?
Bottom line: even in the seemingly “low‑risk” demographic—young, otherwise healthy—Covid can have a genetic secret weapon. Thanks to a little gene called ADAM9, the virus may still hold the upper hand, but scientists are cracking it open, one lab flask at a time.
Pregnant women get sub-par benefit from first vaccine dose
Pregnant and Breast‑Feeding Moms: One Shot Isn’t Enough
New research shows that when moms get their first mRNA Covid‑19 jab while pregnant or nursing, the superhero protection they receive isn’t at its full strength—until they turn in for the second dose.
How the Study was Pitched
- 84 mothers who were pregnant,
- 31 moms who were breastfeeding,
- 16 non‑pregnant, non‑lactating women the same age.
All of them were shot with either Moderna or Pfizer‑BioNTech vaccines. The scientists then measured how their immune systems responded.
What We Learned After the First Injection
- Everyone did hit a spike in antibodies—great start!
- But the spike was noticeably lower for the pregnant and breastfeeding group.
- Other immune markers were also a bit lagging in those two groups.
The team explained that a pregnancy brings huge changes to the body’s defenses. “It’s a lot of immunological juggling to keep the fetus safe,” they wrote.
Why the Second Shot Makes All the Difference
- After the second shot, antibody levels in moms bump back up to the normal range.
- Other markers also “catch up” and level out.
In plain English: if you’re pregnant or nursing, make sure you follow the double‑dose plan. Your body needs the extra boost to keep both you and your little one protected from Covid‑19.
The Bottom Line
Pregnancy reshapes how the immune system reacts to vaccines, so an early first dose is great—but the second shot is the real game‑changer. For those carrying or nursing, staying on schedule isn’t just a recommendation—it’s a crucial step in safeguarding health.
Coronavirus found to infect fat cells
Why Being Overweight Could Be the Dark Side of COVID-19
It’s no secret that people carrying extra weight are at higher risk for a bad bout with COVID‑19. A fresh study sheds a weird new light on why that might be.
Fat Cells: The Unexpected Virus Host
Researchers discovered the SARS‑CoV‑2 virus doesn’t just stick to the lungs—it can actually poke its way into fat tissue. In both lab experiments and post‑mortem examinations of COVID‑19 patients, the virus was found inside two key players:
- Adipocytes – the mature fat cells that store energy
- Macrophages – immune cells that usually keep things in check
The Inflammatory Party That Hits Too Hard
When the virus invades these fat cells, it brings out a fiery inflammatory response. “It’s the kind of immune reaction seen in severe COVID‑19 cases,” explained Dr. Catherine Blish from Stanford University School of Medicine.
What This Means for the Overweight Crowd
Dr. Blish suggests that this pesky infection of fat tissue – and the inflammation it triggers – could be one of the reasons obese individuals suffer more severe outcomes when they contract SARS‑CoV‑2.
Bottom line: if you’ve got a few extra pounds, your body might be doing double time when the virus comes knocking. Stay alert, stay fit, and keep your health plan handy.
